004435 - SOD1-G93A Strain Details
Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text
IJMS | Free Full-Text | Brain Protease Activated Receptor 1 Pathway: A Therapeutic Target in the Superoxide Dismutase 1 (SOD1) Mouse Model of Amyotrophic Lateral Sclerosis
Neuroprotective effects of siRNAp11 in the SOD1-G93A mouse model of... | Download Scientific Diagram
002726 - SOD1-G93A Strain Details
Decreasing SOD1 in muscle does not improve grip strength in SOD1 G93A... | Download Scientific Diagram
Characterization of somatosensory neuron involvement in the SOD1G93A mouse model | Scientific Reports
Neuron-Specific Expression of Mutant Superoxide Dismutase Is Sufficient to Induce Amyotrophic Lateral Sclerosis in Transgenic Mice | Journal of Neuroscience
Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar
IJMS | Free Full-Text | Abnormal Upregulation of GPR17 Receptor Contributes to Oligodendrocyte Dysfunction in SOD1 G93A Mice
A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect
Figure 3.1 from The neuromuscular transmission of the SOD 1 ( G 93 A ) mouse model of Amyotrophic Lateral Sclerosis | Semantic Scholar
Therapeutic effects of immunization with mutant superoxide dismutase in mice models of amyotrophic lateral sclerosis | PNAS
SOD1/Rag2 Mice with Low Copy Number of SOD1 Gene as a New Long-Living Immunodeficient Model of ALS | Scientific Reports
Frontiers | Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis
MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice
Sagittal brain slices from a SOD1 G93A transgenic mouse at terminal... | Download Scientific Diagram
SOD1 aggregation in ALS mice shows simplistic test tube behavior | PNAS
Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology | PNAS
SOD1-G93A Mouse Models - QPS Neuropharmacology
Parkin is a disease modifier in the mutant SOD1 mouse model of ALS. - Abstract - Europe PMC
SOD1-positive aggregate accumulation in the CNS predicts slower disease progression and increased longevity in a mutant SOD1 mouse model of ALS | Scientific Reports
Amyotrophic Lateral Sclerosis disease - QPS Austria
